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  • ABOUT AUTISM
    • What is autism
    • Epidemiology
    • Etiology – causes of the disease
    • Early symptoms
    • Screening
    • Diagnostics
    • What to do after an autism diagnosis ?
      • Early intervention
      • Assisting organizations
      • Where to seek help
      • Collaborations
    • Interventions
      • Applied Behavior Analysis (ABA)
    • Associated medical conditions
    • Prognosis
    • Education of children with autism
    • Families with children with ASD
    • Employment of people with autism
  • RESEARCH
    • Projects
    • Grants
    • Publications
    • Collaborations
    • Cooperation offer
  • SCREENING
    • M-CHAT
  • DIAGNOSTICS
  • EDUCATION
    • ADOS-2 course
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Archives

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  • Archives
    • 1. DECEMBER 2020
    Abnormal neuronal morphology and altered synaptic proteins are restored by oxytocin in autism-related SHANK3 deficient model

    Abnormal neuronal morphology and altered synaptic proteins are restored by oxytocin in autism-related SHANK3 deficient model

    AUTHORS: Reichová, A., Bačová, Z., Bukatová, S., Kokavcová, M., Melišková, V., Frimmel, K., Ostatníková, D. and Bakoš, J. ABSTRACT: Oxytocin has been suggested as a potential therapeutic agent in autism and other neuropsychiatric conditions. Although, the link between the deficit in “SH3 domain and ankyrin repeat containing protein 3” (SHANK3) and autism spectrum disorders is

    Viac
    • Barbara Rašková
    • 2020,
    • Publications
    • 1. APRIL 2018
    Projection length stimulated by oxytocin is modulated by the inhibition of calcium signaling in U-87MG cells

    Projection length stimulated by oxytocin is modulated by the inhibition of calcium signaling in U-87MG cells

    AUTHORS: Zatkova, M., Z. Bacova, F. Puerta, Z. Lestanova, M. Alanazi, A. Kiss, A. Reichova, A.M. Castejon, D. Ostatnikova, and J. Bakos ABSTRACT: Neuropeptide oxytocin contributes to the regulation of glial cell morphology. The precise mechanisms, however, are not yet fully understood. In the present study, we have investigated whether an oxytocin-induced increase of intracellularViac
    • acva
    • 2018,
    • Publications
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