Publications – Page 4 – Akademické centrum výskumu autizmu

- 12. MAY 2022
Improvement of the Clinical and Psychological Profile of Patients with Autism after Methylcobalamin Syrup Administration
AUTHORS: Čorejová, A., Fazekaš, T., Jánošíková, D., Repiský, J., Pospíšilová, V., Miková, M., Rauová, D., Ostatníková, D., Kyselovič, J., Hrabovská, A. ABSTRACT: (1) Background: Autism, also known as autism-spectrum disorder, is a pervasive developmental disorder affecting social skills and psychological status in particular. The complex etiopathogenesis of autism limits efficient therapy, which leads to problems
- Barbara Rašková
- 2022,
- 2022,
- Publications
- Publications
- 22. MARCH 2022
Comparing the impact of the first and second wave of COVID-19 lockdown on Slovak families with typically developing children and children with autism spectrum disorder
AUTHORS: Polónyiová, K., Belica, I., Celušáková, H., Janšáková, K., Kopčíková, M., Szapuová, Ž., Ostatníková, D. ABSTRACT: The aim of this research was to compare the mental health of families with children diagnosed with autism spectrum disorder or typically developing children, during the first and the second wave of COVID-19 outbreak in Slovakia. The study is
- Barbara Rašková
- 2022,
- Publications

- 19. JANUARY 2022
The Relationship of Steroid Hormones, Genes Related to Testosterone Metabolism and Behavior in Boys With Autism in Slovakia
AUTHORS: Lakatošová, S., Janšáková, K., Babková. J., Repiská, G., Belica, I., Vidošovičová, M. and Ostatníková, D. ABSTRACT:Objective: Purpose of the study was to identify the relationship among actual plasmatic levels of steroid hormones and behavioral manifestations in boys with autism and to assess the genetic contribution to these manifestations. Methods: 172 boys with autism under
- Barbara Rašková
- 2022,
- Publications
- 30. DECEMBER 2021
The role of selected postsynaptic scaffolding proteins at glutamatergic synapses in autism-related animal models
AUTHORS: Melišková, V., Havránek, T., Bačová, Z. and Bakoš, J. ABSTRACT: Pathological changes in synapse formation, plasticity and development are caused by altered trafficking and assembly of postsynaptic scaffolding proteins at sites of glutamatergic and gamma-aminobutyric acid synapses, suggesting their involvement in the etiology of neurodevelopmental disorders, including autism. Several autism-related mouse models have been
- Barbara Rašková
- 2021,
- Publications

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